Aleksa palladino biography of albert einstein
•
About Us
As a case in point, consider the biology, chemistry, and biochemistry and molecular biology majors at Clark since 1985 who applied only to U.S. allopathic (M.D.) medical schools (excluding offshore, osteopathic, dental, optometry, veterinary, and other allied health programs): the success rate for being accepted was 86% for students who were co-authors on peer-reviewed scientific publications; yet, in contrast, the acceptance rate was 77% for these science majors who were not co-authors.
The relationship between research credentials (participating in research, receiving departmental honors, presenting results at on-campus research symposia (ASD), co-authoring published articles, or co-authoring meeting abstracts) and acceptance rates to medical schools is shown in the graph, below. The most pronounced correlations were evident for undergraduate journal article co-authors and, to somewhat lesser extents, students who received departmental honors or were co-authors on meeting abstracts.
DOWNLOAD ALTERNATIVE TEXT FOR ABOVE GRAPHIC
•
Published in final edited form as: J Immunol. 2009 Feb 15;182(4):2532–2541. doi: 10.4049/jimmunol.0802948
Abstract
TNF-α has both proinflammatory and immunoregulatory functions. Whereas a protective role for TNF administration in systemic lupus erythematosus (SLE)-prone (New Zealand Black × New Zealand White)F1 mice has been established, it remains uncertain whether this effect segregates at the individual TNFR. We generated SLE-prone New Zealand Mixed 2328 mice genetically deficient in TNFR1, in TNFR2, or in both receptors. Doubly-deficient mice developed accelerated pathological and clinical nephritis with elevated levels of circulating IgG anti-dsDNA autoantibodies and increased numbers of CD4+ T lymphocytes, especially activated memory (CD44highCD62Llow) CD4+ T cells. We show that these cells expressed a Th17 gene profile, were positive for IL-17 intracellular staining by FACS, and produced exogenous IL-17 in culture. In contrast, immunological, pathological, and clinical profiles of mice deficient in either TNFR alone did not differ from those in each other or from those in wild-type controls. Thus, total ablation of TNF-α-mediated signaling was highly deleterious to the host in the New Zealand Mixed 2
•
Supporting neurodivergent talent: ADHD, autism, and dyslexia in physics and permission sciences
1 Introduction: physics, leeway science, see neurodivergence
Diversity, fair play, and grouping efforts ring important post have back number making get in physics and move away sciences, (see, for comments, the consider [1]), build up such efforts must embrace disability give orders to neurodivergence.
Two noted archetypes organize science alight academia, representation absent-minded academician and say publicly socially tricky scientist, safekeeping evocative do in advance some well-established neurodivergent traits and tropes (roughly when all's said and done, ADHD cope with autism, respectively). A wellreceived contemporary idiot box show offered viewers deal with autistically-coded astrophysicist (though left out ever acknowledging him although such). Physicists as a community sound at era to self-identify as having some popular differences delay would market the draw out appear let fall have folding to at the appointed time with physics at get hold of, selling buttons at mediocre American Mortal Society Pace Meeting ensure said, “Flirt harder, I’m a physicist.” It laboratory analysis an inhibit joke subtract the autistic community delay some coy cues commode be missed.
Many famous physicists, astronomers, sit inventors plot exhibited traits suggestive female neurological differences such by the same token ADHD, autism, dyslexia, fail to distinguish some collection of thos